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MySQL Error: 1194 (Table 'pwn_comment' is marked as crashed and should be repaired)
#0 dbbase_sql->halt(Invalid SQL: select count(id) from pwn_comment where pid='611256' and iffb='1') called at [D:\wwwroot\hs21cn2043\wwwroot\includes\db.inc.php:54] #1 dbbase_sql->query(select count(id) from {P}_comment where pid='611256' and iffb='1') called at [D:\wwwroot\hs21cn2043\wwwroot\comment\module\CommentContent.php:65] #2 CommentContent() called at [D:\wwwroot\hs21cn2043\wwwroot\includes\common.inc.php:551] #3 printpage() called at [D:\wwwroot\hs21cn2043\wwwroot\comment\html\index.php:13]
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Warning: mysql_query() [function.mysql-query]: Unable to save result set in D:\wwwroot\hs21cn2043\wwwroot\includes\db.inc.php on line 50
Database error: Invalid SQL: select * from pwn_comment where pid='611256' and iffb='1' order by id limit 0,10
MySQL Error: 1194 (Table 'pwn_comment' is marked as crashed and should be repaired)
#0 dbbase_sql->halt(Invalid SQL: select * from pwn_comment where pid='611256' and iffb='1' order by id limit 0,10) called at [D:\wwwroot\hs21cn2043\wwwroot\includes\db.inc.php:54] #1 dbbase_sql->query(select * from {P}_comment where pid='611256' and iffb='1' order by id limit 0,10) called at [D:\wwwroot\hs21cn2043\wwwroot\comment\module\CommentContent.php:167] #2 CommentContent() called at [D:\wwwroot\hs21cn2043\wwwroot\includes\common.inc.php:551] #3 printpage() called at [D:\wwwroot\hs21cn2043\wwwroot\comment\html\index.php:13]
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网友点评-A. Thornberry. 1998. Inhibition of human caspases by peptide-based and macromolecular inhibitors.-线缆测高仪,超声波测高仪, 手持式测高仪-上海交通大学科技园上海野豹企业发展公司
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A. Thornberry. 1998. Inhibition of human caspases by peptide-based and macromolecular inhibitors.
A. Harrison, E. Altman, D. M. Lublin, and K. Dimmock. 1996. The HeLa cell receptor for enterovirus 70 is decayaccelerating factor (CD55). J. Virol. 70:5143?152. 18. Kinoshita, T., M. E. Medof, R. Silber, and V. Nussenzweig. 1985. Distribution of decay accelerating aspect in the peripheral blood of normal individ-
JOURNAL OF VIROLOGY, Sept. 1998, p. 7420?427 0022-538X/98/ 04.00 0 Copyright ?1998, American Society for Microbiology. All Rights Reserved.Vol. 72, No.Measles Virus Spread and Pathogenesis in Genetically Modified Mice?BRANKA MRKIC,1 JOVAN PAVLOVIC,2 THOMAS RULICKE,three PIETRO VOLPE,1 CHRISTIAN J. BUCHHOLZ,1 DENNIS HOURCADE,4 JOHN P. ATKINSON,4 ADRIANO AGUZZI,five AND ROBERTO CATTANEO1* ?Institut fur Molekularbiologie Abt. I,1 Institut fur Medizinische Virologie,two Biologisches Zentrallabor,3 ?and Institut fur Neuropathologie,five Universitat Zurich, Zurich, Switzerland, and ????Division of Rheumatology, Division of Internal Medicine, Washington University School of Medicine, St. Louis, MissouriReceived two March 1998/Accepted 8 JuneAttenuated Edmonston measles virus (MV-Edm) will not be pathogenic in typical mice. We show here that MVEdm inoculated via the natural respiratory route has a limited propagation within the lungs of mice using a targeted mutation inactivating the alpha/beta interferon receptor. A high dose of MV-Edm administered intracerebrally is SN-38Protocol lethal for about half of those mice. To study the consequences with the availability of a high-affinity receptor for MV propagation, we generated alpha/beta interferon-defective mice expressing human CD46 with humanlike tissue specificity. Intranasal infection of those mice with MV-Edm resulted in enhanced spread for the lungs and more prominent inflammatory response. Virus replication was also detected in peripheral blood mononuclear cells, the spleen, and also the liver. In addition, intracerebral inoculation of adult animals with low MV-Edm doses triggered encephalitis with pretty much inevitably lethal outcome. We conclude that in mice alpha/beta interferon controls MV infection and that a high-affinity receptor facilitates, but is not strictly required for, MV spread and pathogenesis. Measles remains one of several major causes of infant death in developing nations (40) and, in uncommon situations, persistent measles virus (MV) infection induces the lethal neurodegenerative illness subacute sclerosing panencephalitis (SSPE) (10, 55). Direct studies PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/28144193 of early MV replication in humans are lacking, but experimental studies in monkeys (32, 58) and histopathological observations in humans (33) suggest neighborhood replication in the respiratory mucosa a few days Oprozomib In Vivo following infection. MV may perhaps then spread, possibly carried in pulmonary macrophages, to draining lymph nodes and from there enter the bloodstream carried in leukocytes, disseminating first to lymphoid tissues and then to tissues throughout the body (40). MV infection of adult rodents is restricted to brain-adapted strains inoculated intracerebrally (29).A. Thornberry. 1998. Inhibition of human caspases by peptide-based and macromolecular inhibitors.
A. Thornberry. 1998. Inhibition of human caspases by peptide-based PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/23770981 and macromolecular inhibitors. J. Biol. Chem. 273:32608?2613. 16. Greve, J. M., G. Davis, A. M. Meyer, C. P. Forte, S. Connolly Yost, C. W. Marlor, M. E. Kamark, and a. McClelland. 1989. The important human rhinovirus receptor is ICAM-1. Cell 56:839?45.
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